品牌分类
品牌咨询
- Electrochromic Polymers Grown on Nanotemplat
- i百联iphone为什么便宜
- 丰都县3个乡镇卫生院数字化医疗设备(包二) 第二次(19A0005)采购...
- IBL International GmbHELISA试剂盒
- 试剂IBL International代理
- Tecan透明滤芯吸头1000ul 盒装灭菌自动化吸头
- IBL唾液褪黑激素检测试剂_价格厂家供应商_深圳市科润达 ...
- 常用血清学检测方法介绍 QQ
- 怎样用酶标仪(光度测定法)做内毒素检测资料资讯...
- 中国首家“企业家功能医学抗衰老俱乐部”面世榕城
- Tecan全国代理_Tecan授权代理,Tecan全国代理,北京百奥创新,Tecan...
- IBL 品牌介绍及代理商/经销商名单品牌专区
联系方式
公司地址
苏州工业园区生物纳米园A4#216
联系电话
4000-520-616 / 18915418616
传真号码
0512-67156496
电子邮箱
info@ebiomall.com
公司网址
https://www.ebiomall.com
商品描述
| Quantity/Tests | 50µg |
| Species/Antigen | SyntheticpeptideofhumanAmyloid-beta(38-42)(GVVIA) |
| Host | Mouse |
| Subclass/Clone | IgG2b |
| Type | Monoclonal |
| Purification | ProteinA |
| Label | No |
| RegulatoryStatus: | Forresearchuseonly |
Detailsfor:Anti-HumanAmyloid-beta(38-42)(44A3)MouseIgG
iBL的Tecan由四名工程师于1980年在瑞士Hombrechtikon村成立，旨在开发测量，分析和实验室自动化设备。最初的办公室位于经过翻新的农舍中，这是首席执行官兼创始人亨氏·阿普兰纳普（Heinz Abplanalp）的家庭住宅。 "Originalarticle#11115
Alzheimer"sdisease(AD)ischaracterizedbythepresenceofextracellularplaquesandintracellularneurofibrillarytangles(NFTs)inthebrain.Themajorproteincomponentoftheseplaquesisbetaamyloid(Aβ)peptide,a40to43aminoacidpeptidecleavedfromamyloidprecursorproteinbyβ-secretaseandγ-secretase.IncreasedreleaseofAβ42orAβ43,bothofwhichexibitagreatertendencytoaggregatethanAβ40,occursinindividualsexpressingcertaingeneticmutations,ApoEallelesormayinvolveotherundiscoveredfactors.ManyresearcherstheorizethatitisthisincreasedreleaseofAβ42/Aβ43whichleadstotheabnormaldepositionofAβandtheassociatedneurotoxicityinthebrainsofaffectedindividuals."
ForconcretedatapleaseconsulttheInstructionforUseinthedownloadboxontherightside.Alzheimer"sdisease(AD)ischaracterizedbythepresenceofextracellularplaquesandintracellularneurofibrillarytangles(NFTs)inthebrain.Themajorproteincomponentoftheseplaquesisbetaamyloid(Aβ)peptide,a40to43aminoacidpeptidecleavedfromamyloidprecursorproteinbyβ-secretaseandγ-secretase.IncreasedreleaseofAβ42orAβ43,bothofwhichexibitagreatertendencytoaggregatethanAβ40,occursinindividualsexpressingcertaingeneticmutations,ApoEallelesormayinvolveotherundiscoveredfactors.ManyresearcherstheorizethatitisthisincreasedreleaseofAβ42/Aβ43whichleadstotheabnormaldepositionofAβandtheassociatedneurotoxicityinthebrainsofaffectedindividuals."
